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BPC-157 A 2024 systematic review in HSS Journal (Vasireddi et al.) identified 36 included studies 35 preclinical, 1 small clinical across nearly three decades
Furthermore, our data suggest potent inhibition of the FAT helix 2-3 site is sufficient for cellular disruption of the FAK-paxillin interaction, which is in alignment with previous reports characterizing the interaction 25
This can minimize the risk of adverse reactions and maximize the efficacy of the treatment
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