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Conversely, studies have shown that CXCL10-neutralizing antibody treatment blocks CD8 + T cell skin recruitment, significantly inhibiting disease progression in vitiligo mouse models ( + T cells to the epidermis, resulting in melanocyte damage in vitiligo ( Moreover, CCL5 (known as RANTES) plays a significant role in the establishment and maintenance of the inflammatory microenvironment in vitiligo by chemotactically attracting monocytes and macrophages via its receptor CCR5 ( + T cells to target melanocytes within the epidermis
However, it was shown that GSH depletion induces ferroptosis in some cell types but not others 23
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