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however, the clinical manifestations and immunological phenotypes of diseases related to the PI3K/AKT/mTOR pathway are diverse [1]

Thus, hyperglycemia may increase responsiveness to local low-grade inflammation 16,17

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Since co-treatment of UK-5099 and JMF4073 depleted dTTP pool and suppressed the growth of T315-Bcr-Abl-32D cells, we proposed that reducing GSH biosynthesis induces not only oxidative stress but also replication stress in tumor cells, which demands the synthesis of dTTP and dCTP for survival